About Our Lab
Our research focuses on the basal ganglia, a group of subcortical brain nuclei that are critical for voluntary movement, learning and motivation, and the primary site of dysfunction in psychomotor disorders such as Parkinson's disease, Huntington's disease, obsessive-compulsive disorder and addiction.
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Selected Publications
Lahiri AK, Bevan MD.
Dopaminergic Transmission Rapidly and Persistently Enhances Excitability of D1 Receptor-Expressing Striatal Projection Neurons. 2020 Neuron 106:277-290
McIver EL, Atherton JF, Chu HY, Cosgrove KE, Kondapalli J, Wokosin D, Surmeier DJ, Bevan MD.
Maladaptive Downregulation of Autonomous Subthalamic Nucleus Activity Following the Loss of Midbrain Dopamine Neurons. 2019 Cell Reports 28:922-1002
Chu HY, McIver EL, Kovaleski RF, Atherton JF, Bevan MD.
Loss of Hyperdirect Pathway Cortico-Subthalamic Inputs Following Degeneration of Midbrain Dopamine Neurons.
2017 Neuron 95:1306-1318
Atherton JF, McIver EL, Mullen MRM, Wokosin DL, Surmeier DJ, Bevan MD.
Early dysfunction and progressive degeneration of the subthalamic nucleus in mouse models of Huntington's disease.
2016 eLife 5:e21616
Chu HY, Atherton JF, Wokosin D, Surmeier DJ, Bevan MD.
Heterosynaptic regulation of external globus pallidus inputs to the subthalamic nucleus by the motor cortex.
2015 Neuron 85: 364–376